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Irritable Bowl Syndrome is a result of a dysfunction of the nerves that control the muscles of the gastrointestinal tract. However, the nervous system of the intestines is very complex.
Nerves can be found along the entire span of the gastrointestinal tract that begins at the esophagus and ends at the anus. This network of nerves interacts with the many nerves associated with the spinal cord. The nerves contained in the spinal cord then interact with the nerves of the brain. (The gastrointestinal tract is exceeded by the amount of nerves only by the brain and spinal cord.) Therefore, it is possible for irritable bowl syndrome to cause the nerves to malfunction in the spine, the brain as well as the intestinal tract.
The nervous system that controls the gastrointestinal organs, as with most other organs, contains both sensory and motor nerves. The sensory nerves continuously sense what is happening within the organ and relay this information to nerves in the organ’s wall. From there, information can be relayed to the spinal cord and brain.
The sensory input results in the motor responses called contractions and relaxation in the muscles of the organ. As such, abnormal inputs to the sensory nerves or the motor nerves or even at the receptors in the intestines, brain and spinal cord can produce irritable bowl syndrome.
Some researchers argue that the cause of functional diseases is abnormalities in the function of the sensory nerves. For example, normal activities, such as stretching of the small intestine by food, may give rise to abnormal sensory signals that are sent to the spinal cord and brain, where they are perceived as pain.
Other researchers argue that the cause of functional diseases is abnormalities in the function of the motor nerves. For example, abnormal commands through the motor nerves might produce a painful spasm (contraction) of the muscles. Still others may be due to abnormalities within the processing centers.
Lastly, one area that is receiving a great deal of scientific attention is the potential role of gas produced by intestinal bacteria in patients with IBS. Studies have demonstrated that patients with irritable bowl syndrome produce larger amounts of gas than individuals without irritable bowl syndrome, and the gas may be retained longer in the small intestine.
If was found that the gas caused an increase in abdominal size over the day with the size being the largest in the evening then going back to normal the following day. This condition is generally caused by poor digestion in conjunction with the absorption of sugars. Poor absorption of lactose and fructose in combination with poor digestion will aggravate irritable bowl syndrome due to the increase in production of gas.